Cercetătorii francezi au demonstrat interacţiunea dintre două proteine din creier, proteina FKBP52 şi proteina Tau, ale căror anomalii intervin în mecanismul a numeroase patologii neurodegenerative, printre care şi maladia Alzheimer.
Prezentă în mod natural în creier, forma normală a proteinei Tau asigură buna funcţionare a neuronilor. Forma anormală a proteinei Tau este însă un indice al maladiei Alzheimer.
Cercetătorii au demonstrat în laborator că o cantitate mare de proteină FKBP52, împiedică acumularea proteinei Tau în celulele nervoase. Ideea cercetătorilor francezi este de a folosi proteina FKBP52 pentru a inhiba activitatea proteinei Tau în forma sa anormală, în particular prin protejarea mecanismului de hrănire al neuronilor.
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